Spinal Stenosis
I. Background
Spinal
stenosis is a pathologic condition common in people over 50
years of age. It is caused by narrowing of the spinal canal
and/or the neural foramina roots, which produces abnormal pressure
on spinal nerve roots or the blood vessels supplying the nerve
roots. The condition may be congenital (achondroplasia) or
acquired. Acquired causes include degenerative changes of the
spine, ie., disk degeneration, facet joint hypertrophy,
ligamentous calcification and vertebral osteophytes;
spondylolisthesis or other malalignment; progressive enlargement
of the fusion mass after a posterior spinal fusion; Paget's
disease or fluorosis. It may also occur after an unreduced burst
fracture or fracture-dislocation of the lumbar spine. Patient
population at risk varies from young adults (achondroplasia or
spinal fractures), to middle aged patients with a single nerve
root trapped by lateral recess stenosis to elderly patients with
multilevel central and lateral stenosis. Back pain may or may
not be a presenting complaint. Sciatica or claudication in one
or both legs are common presenting complaints. Pain, sensory
disturbance, and motor weakness experienced by these patients
are often aggravated by exercise and relieved by bending forward
or flexing the low back by sitting. Bowel and/or bladder
dysfunction and other neurologic symptoms may occur.
II.
Diagnostic Criteria
A.
Pertinent Historical and Physical Findings
Patients
may exhibit few objective findings such as reflex changes, motor
weakness, sensory deficits, or inability to stoop.
Hyperextension of the spine is usually limited and painful. A
hallmark of this condition is that the severity of the clinical
symptoms change with the position of the patient. Lower
extremity reflexes often disappear, and pain, numbness, and
weakness in the legs may appear after exercise. Sciatic tension
tests are usually negative but weakness of the extensor hallucis
longus muscle may be present. The condition may progress to the
point of greatly impairing the patient's quality of life. At present, there are no studies that correlate the degree
of spinal compression with the severity of symptoms.
B.
Appropriate Diagnostic Tests and Examinations (one or
more)
1.
Routine
x-rays of the low back will not confirm diagnosis but are needed
to rule out other conditions.
2.
Myelography
may confirm diagnosis
3.
CT
scan may confirm diagnosis
4.
MRI
may confirm diagnosis
5.
Bone
scan
6.
Cystometrogram
if symptoms warrant
7.
Evaluation
of vascular system if symptoms warrant
C. Inappropriate Diagnostic Tests and Examinations
1.
Routine
x-rays of the low back as sole diagnostic test
2.
EMG
as sole diagnostic test
3.
Thermography
4.
Ultrasound
5.
Bone
scan as sole diagnostic test
D. Exceptions to Above
Criteria
1.
EMG
in conjunction with neurologic consultation may be useful in
preoperative planning for delineating involved nerve roots'.'
2.
Bone
scans, laboratory tests, and other studies may be indicated in
the evaluation of selected patients.
E. Evolving Diagnostic
Tests and Examinations
1.
Ambulatory performance of myelography is an evolving outpatient
technique
F.
Supporting Evidence
Studies have documented that myelography with water
soluble contrast media and CT
scans are the most accurate tests for con-firming the
diagnosis of spinal stenosis. However, of these two studies,
myelography is more accurate for documenting central stenosis
and CT scans are more accurate for documenting nerve root
canal stenosis. For this reason, most orthopaedists order both
tests. EMG findings are abnormal in 80% of patients with
this disorder, but the findings lack specificity, which make EMG
an inappropriate test as the sole modality for confirming the
diagnosis.
III.
Treatment
A.
Outpatient Treatment
1.
Nonoperative Treatment
a.
Indications: absence of severe or progressively abnormal
neurologic signs
b.
Treatment Options
1)
Restricted
activity
2)
Ambulatory
rest
3)
Physical
modalities
4)
Nonsteroidal
anti-inflammatory drugs
5)
Analgesics
6)
Periodic,
epidural steroid injections
7)
Spinal
orthotic supportive devices
c.
Home Health Care: monitor compliance with treatment protocol and
alter as indicated.
d.
Rehabilitation
1)
Spinal
muscle strengthening exercises
2)
Endurance
and strength reactivation programs
e.
Supporting Evidence: A few studies exist to show the value of
short periods of bed rest followed by con-trolled activity in
relieving low back pain and sciatica. These studies are mostly
clinical series, not prospective controlled studies. No data
currently exist to indicate the percent of patients who will
need surgical intervention or
the percent who may function satisfactorily without
treatment.
2.
Ambulatory Surgery, Indications: none
B.
Inpatient Treatment
1.
Nonoperative Treatment
a.
Indications for Admission
1)
Severe
back and leg pain unresponsive to outpatient treatment
2)
Myelogram
or combination myelogram
and
CT scan
b.
Treatment Options
1)
Monitored
bed rest with sedation and
analgesics
during acute phase
2)
Epidural
steroid injection(s)
3)
Flexion
body cast, bracing
4)
Endurance
and strength reactivation
c.
Indications for Discharge
1)
Relief
of back and/or leg pain
2)
No
response to nonoperative treatment options pending patient's
consideration of surgical intervention
d.
Home Health Care: Monitor compliance with treatment protocol and
alter as indicated
e.
Rehabilitation
1)
Spinal
muscle strengthening exercises
2)
Endurance
and strength reactivation programs
2.
Operative Treatment
a.
Indications for admission
1)
Quality
of life significantly impaired
2)
Failure
of nonoperative treatment to relieve symptoms
3)
Presence
of severe or progressive abnormal
neurologic
signs
4)
Diagnosis
confirmed by myelography, CT scan,
or
MRI
b.
Procedure Options
1)
Decompression
2)
Decompression
with fusion
3)
Decompression
with fusion and instrumentation
c.
Indications for Discharge
1) Uncomplicated
a)
Approximately
5 to 10 days following laminectomy
b)
Approximately
7 to 15 days if spinal fusion performed in conjunction with
laminectomy
2)
Complicated: stay may be extended for co-morbidity (e.g.,wound
infection,thrombophlebitis, durotomy or spinal fluid leak).
d.
Home Treatment
1)
Visiting
nurse and/or physical therapist may be required
to assist patient in returning to activities of daily
living.
2)
Patient
may need back brace.
e.
Rehabilitation
1)
Assistance
with spinal muscle strengthening program may be needed.
2)
Overall
general physical conditioning (e.g.,
cardiovascular and pulmonary
fitness
activities)
f.
Supporting Evidence: Surgical intervention in spinal stenosis
remains an elective decision based on the surgeon's clinical
judgment and the patient's personal assessment of his/her
quality of life impairment. The preferred surgical procedure for
spinal stenosis remains undecided although most authorities
favor decompressive
laminectomy without fusion in central spinal stenosis.
Lateral recess decompression may create instability or potential
instability requiring fusion. Fusion may also be indicated in addition to the
laminectomy in patients with symptomatic degenerative
spondylolisthesis or other forms of spinal instability.
Satisfactory results from surgery can be expected in
approximately 85% of patients. Long-standing nerve root
compression may result in a less satisfactory surgical result.
C. Inappropriate
Treatment
1.
Chemonucleolysis
2.
Percutaneous
diskectomies
3.
Spinal
fusion without decompressive laminectomy
D. Exceptions to Above
Criteria: None
E. Estimated Duration of
Care
1.
Nonoperative
treatment indeterminant based on patient's symptoms
2.
Operative
treatment 6 to 12 months
F. Anticipated Outcomes
1.
Complete
or partial relief of back and leg pain
2.
Improved
quality of life
G. Evolving Therapeutic
Procedures
1. Laminoplasty or interlaminar decompression
H. Modifiers (age, sex, and co-morbidity)
Patients
with spinal stenosis caused by spinal trauma or achondroplasia may
need surgical treatment at an earlier age. When surgery is
required in patients with achondroplasia, laminectomy without
fusion is usually sufficient. Myelogram may be done through an
upper cervical puncture in patients with achondroplasia to avoid
causing neurologic deficits.
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Traverse City, Michigan-MI, USA 49684
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